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    • 1. 实用新型
    • 油冷器
    • TWM290924U
    • 2006-05-21
    • TW094220578
    • 2005-11-28
    • 坤典利有限公司 KUNG DEAN LI CO., LTD.
    • 吳文忠 WU, WEN CHUNG
    • F01P
    • 一種油冷器,係包括有進油槽、複數管體、散熱片及出油槽,其中,將潤滑油經由進油管路進入進油槽之噴油元件,藉由噴油元件霧狀噴出潤滑油,分散油溫後,該潤滑油會經由進油槽沿著複數管體之管壁往下流進出油槽以供強力分散油溫,再藉由複數管體等距離之散熱片分散油溫,以及藉由外來的風力或使用者本身的散熱風扇之風力,使風力經由散熱片外圍之導流板進入,該風力在複數管體裡產生漩渦式風向,可做為強而有力之散熱作用,以達到降溫效果。
    • 一种油冷器,系包括有进油槽、复数管体、散热片及出油槽,其中,将润滑油经由进油管路进入进油槽之喷油组件,借由喷油组件雾状喷出润滑油,分散油温后,该润滑油会经由进油槽沿着复数管体之管壁往下流进出油槽以供强力分散油温,再借由复数管体等距离之散热片分散油温,以及借由外来的风力或用户本身的散热风扇之风力,使风力经由散热片外围之导流板进入,该风力在复数管体里产生漩涡式风向,可做为强而有力之散热作用,以达到降温效果。
    • 4. 发明授权
    • Compositions and methods for inhibiting vascular permeability
    • 抑制血管通透性的组合物和方法
    • US08309519B2
    • 2012-11-13
    • US13111776
    • 2011-05-19
    • Dean LiChristopher JonesNyall London
    • Dean LiChristopher JonesNyall London
    • A61K38/16A61K38/17C07K14/435
    • A61K38/17A61K38/00C07K14/70503G01N33/5023G01N2800/164
    • Compounds, compositions and methods for inhibiting vascular permeability and pathologic angiogenesis are described herein. Methods for producing and screening compounds and compositions capable of inhibiting vascular permeability and pathologic angiogenesis are also described herein. Pharmaceutical compositions are included in the compositions described herein. The compositions described herein are useful in, for example, methods of inhibiting vascular permeability and pathologic angiogenesis, including methods of inhibiting vascular permeability and pathologic angiogenesis induced by specific angiogenic, permeability and inflammatory factors, such as, for example VEGF, bFGF and thrombin. Methods for treating specific diseases and conditions are also provided herein.
    • 本文描述了用于抑制血管通透性和病理性血管生成的化合物,组合物和方法。 本文还描述了生产和筛选能够抑制血管通透性和病理性血管生成的化合物和组合物的方法。 药物组合物包括在本文所述的组合物中。 本文描述的组合物可用于例如抑制血管通透性和病理性血管生成的方法,包括抑制由特异性血管生成,通透性和炎性因子(例如VEGF,bFGF和凝血酶)诱导的血管通透性和病理性血管发生的方法。 本文还提供了治疗特定疾病和病症的方法。
    • 8. 发明授权
    • Method for etching silicon oxynitride and dielectric antireflection coatings
    • 蚀刻氮氧化硅和介电抗反射涂层的方法
    • US06291356B1
    • 2001-09-18
    • US09317655
    • 1999-05-24
    • Pavel IonovSung Ho KimDean LiChun YanJames Chang Wang
    • Pavel IonovSung Ho KimDean LiChun YanJames Chang Wang
    • H01L21302
    • H01L21/32139H01L21/0276H01L21/31116H01L21/3143
    • The present disclosure pertains to a method for plasma etching a semiconductor film stack. The film stack includes at least one layer comprising silicon oxynitride. The method includes etching the silicon oxynitride-comprising layer using an etchant gas mixture comprising chlorine and at least one compound containing fluorine and carbon. The atomic ratio of fluorine to chlorine in the etchant gas ranges between about 3:1 and about 0.01:1; preferably, between about 0.5:1 and about 0.01:1; most preferably, between about 0.25:1 and about 0.1:1. The etchant gas forms a fluorine-comprising polymer or species which deposits on exposed surfaces adjacent to the silicon oxynitride-comprising layer in an amount sufficient to reduce the etch rate of an adjacent material (such as a photoresist) while permitting the etching of the silicon oxynitride-comprising layer.
    • 本公开涉及用于等离子体蚀刻半导体膜堆叠的方法。 薄膜叠层包括至少一层包含氮氧化硅的层。 该方法包括使用包含氯和至少一种含氟和碳的化合物的蚀刻剂气体混合物来蚀刻含氧氮化硅层。 蚀刻剂气体中氟与氯的原子比范围为约3:1至约0.01:1; 优选约0.5:1至约0.01:1; 最优选在约0.25:1至约0.1:1之间。 蚀刻剂气体形成含氟聚合物或物质,它们以足以降低邻近材料(例如光致抗蚀剂)蚀刻速率的量沉积在与含氮氧化硅层相邻的暴露表面上,同时允许蚀刻硅 含氧氮化物的层。
    • 10. 发明申请
    • Mice which are +/- or -/- for the elastin gene as models for vascular disease
    • 作为血管疾病模型的弹性蛋白基因的+/-或 - / - 小鼠
    • US20050223419A1
    • 2005-10-06
    • US11046006
    • 2005-01-28
    • Mark KeatingDean Li
    • Mark KeatingDean Li
    • A61K38/39A61L31/04A61P9/10A01K67/027A61K38/54C12N5/06
    • A61L31/047A61K38/39A61L31/043
    • Elastin, the main component of arterial extracellular matrix, was thought to have a purely structural role. Consistent with this view, elastin hemizygous mice maintain arterial extensibility by increasing the number of elastic lamellae during development. However, mice lacking elastin die of obstructive arterial pathology. This pathology results from subendothelial proliferation and reorganization of smooth muscle, cellular changes similar to those observed in atherosclerosis. Thus, elastin is a molecular determinant of arterial morphogenesis and likely plays a central role in vascular disease. Mice which are heterozygous and null for the elastin gene have been developed. These mice are extremely useful for screening for drugs useful for treating persons with atherosclerosis, hypertension, SVAS or other vascular diseases.
    • 动弹性细胞外基质的主要成分弹性蛋白被认为具有纯粹的结构作用。 与此观点一致,弹性蛋白半合子小鼠通过在发育过程中增加弹性薄片的数量来维持动脉的伸展性。 然而,缺乏阻塞性动脉病理学弹性蛋白酶的小鼠。 这种病理学结果来自内皮细胞增殖和平滑肌重组,细胞变化与动脉粥样硬化中观察到的细胞变化相似。 因此,弹性蛋白是动脉形态发生的分子决定因素,并且可能在血管疾病中起核心作用。 已经开发了对于弹性蛋白基因是杂合的和无效的小鼠。 这些小鼠对于筛选可用于治疗动脉粥样硬化,高血压,SVAS或其他血管疾病的药物非常有用。